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project:brain_hacking:gmi [2012/12/03 22:15]
project:brain_hacking:gmi [2014/08/24 14:55] (current)
ata Experience update
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 ====== Graphic mind interface ====== ====== Graphic mind interface ======
 +<note important>Experience has shown that experimentation in this area may be dangerous. May lead to changes in the perception of reality and symptoms of "schizophrenia". 
 ===== Goals: ===== ===== Goals: =====
-Final goal : Graphic interface to mind based on subconsciousnes generated visul data showed in visual field ( mind eye ).+Final goal : Graphic interface to mind based on subconsciousnes generated visual data showed in visual field ( mind eye ). 
 +===== Experience ===== 
 +Experience over the last two years have shown many risks,side effects and lot of ways how not to do it. 
 +Intention make this interface cause many poroblems its create intention/suggestions working in the subconscious which i long time  
 +not knew about.This suggestions cuase lowering of preception priority of physical senses and lowering significance of physical reality as a whole. 
 +These changes did not create the required interface (for reasons which i found out much later)but create interesting side effects. 
 +Lowering priority of physical reality freed resources for this perception used , this free resources began to be used subconsciousnes processes. 
 +Physical manifestations were more fatique and impaired senses especially vision. Mental symptoms increased perception of emotions, thoughts and emotions  
 +foreign to me, preception of "ghosts", comunication with "ghosts" and different kinds of aspects of personality, better comunication with awareness. 
 +Especially in the beginning it was very difficult i had headaches and emotions with  limited control over them.Gradually I learned how to navigate  
 +and working with them sensations became intense but controlable.I knew that essentially exhibit classic symptoms of schizophrenia but  
 +i began have state sufficiently under control to allow me to explore this state and its mechanisms deeper (dangerous but exciting journey). 
 +I have gained an interesting new perspective on how schizophrenia works which included a much broader view of different interactions at different levels. 
 +For example, as trigger "schizophrenia" can serve any phenomenon that reduces priority physicals or physiacal reality importance or something witch  
 +increse priority of nonplysical senses and reality.This lowering can by cased for exmple by depresion , deterioration of the physical senses , intention/suggestions. 
 +Recently, trying to fix the setting of priorities of physical senses and at the same time you maitain advantages nonphysical perception give me. 
 ===== Notes ===== ===== Notes =====
 ==== Hypangogic state ==== ==== Hypangogic state ====
 +<note>idea: comaparation of automatic drawing and EEG data </note>
 +  *12-14Hz EEG spindles during the SO transition , but may reflect loss of consciousness of the external environment.
 +  *Adiminution in the N3 ERP component may also index an attentional shift from external sensation tointernal imagery
 +  *Occipital alphablocking with low-voltage, 4-7 Hz theta waves has also beenreported that appears to be associated with high rates of imagery recall 
 +  *EEG-derived dipoles indicate that spontaneous thoughts at SO differin localization (anterior vs. posterior) and frequency band(delta/theta and alpha primarily) depending upon the specificcontent under investigation (e.g., abstract vs. visual)
 +  *Dream-like experiencesoccur most frequently in SO-stage 4 (flattening of the EEG to less than 20µV)
 +  *{{http://i27.tinypic.com/51bnep.png}}
 +  *frequency of kinesthetic images peaks in SO-stage 1 thendecreases steadily in later stages, whereas the opposite is truefor visual images
 +  * Distribution of Modalities OverSO Stages
 +Of the 120 images, 80.9% (97) were classified as occurringin SO-stage 4 (EEG flattening) whereas 12.5% (15) wereclassified as occurring in SO-stage 5 (theta ripples). Onlyseven images (5.8%) occurred in SO-stages 1 or 2, a singleimage (0.8%) occurred in SO-stage 7, and no images occurredin SO-stages 8 and 9.
 +* Spectral Powerof Kinesthetic and Visual Images
 +  *1) awake EEG samples, forbfrequencies faster than delta, had higher power than did corresponding image samples–regardless of whether thesewere kinesthetic or visual samples;
 +  *2) image samples had higher delta power than awake samples;
 +*indicated reduced power during image samples regardless of whether these were visual or kinesthetic. The effect was by farmost generalized for alpha, for which all 19 electrodessignificantly discriminated awake from imagery samples.
 +*Findings from two previous self-observational studies(Nielsen, 1991, 1995) suggested that images produced withthis method are temporally-linked to the initiation of muscleatonia and/or phasic neuromuscular events such as limbtwitches and head jerks. If this is the case, then it is also likelythat atonia-linked movements experienced by our subjectsduring the task awakened them before they progressed beyondSO-stage 4 or, at most, 5.
 +<note>measure delta increase Fp1(kinesthetic) T5(visual) </note> 
 ==== Ganzfeld  ====  ==== Ganzfeld  ==== 
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 and with earlier findings of other authors mentioned above and with earlier findings of other authors mentioned above
 +*comparing electrophysiological
 +signatures of sleep onset, waking mentation and ganzfeld,
 +it was established that ganzfeld imagery occurs in a brain
 +functional state which is distinctly different from that
 +occurring at sleep onset
 +The subjects’ eyes
 +were covered by translucent, anatomically shaped goggles,
 +which were illuminated by bright red light. Simultaneously they
 +were listening to the monotonous sound of a waterfall through
 +headphones. Sound intensity was individually adjusted to a
 +subjectively comfortable level. This procedure has proven to
 +accomplish an almost perfect homogenisation of visual and
 +acoustic sensory field
 +*alfa1/alfa2 location Pz
 +*a1 power shows a minor but continuous decrease during the entire 30-s analysis interval,while a2 displays a distinct tri-phasic time course: initially (30
 +to 21 s) reduced power, relative to baseline level; followed by a
 +F burst_up to the baseline level in the second sub-interval (20 to11 s), and finally a monotonic and strong decrease during thelast sub-interval (10 to 2 s). These time courses are reflected by the corresponding Z values
 +*differences in the time course of the two
 +a sub-bands are particularly well expressed by the a2/a1 ratio ,  8–10 Hz (a1), 10–12 Hz (a2)  (a2/a1=-2)
 +*negative coreleation of a2 power
 +*There was a positive global correlation for variables b3 power and vividness of imagery 
 ==== Gamma /theta synchrony  ==== ==== Gamma /theta synchrony  ====
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 {{project:brain_hacking:attentionfocusproblem.jpg}} {{project:brain_hacking:attentionfocusproblem.jpg}}
 +====31.5 update ====
 +EOG and automatic drawing test 10min 
 +  *Automatic drawing of FV and NV
 +  *horizontal EOG
 +Goal : compare EOG (delta amplitude ) to increase FV priority
 +Result : 
 +  *all FV priority peaks  found in EOG (+-5s drawing is not that precize ,recording about 9min )  
 +  * few peaks in EOG but not in drawing --) only in pars where priority is high and eye movemt or focus to FV no longer incerease it   OK
 +Audio EOG biofeedback to minimalize eye movements 
 +===Pasive realx + sugestion not to move eyes and breathing to eyes about 1 min  ===
 +    * preception priority P (0-5, lower have higher priority )
 +    * NV non-physical vision 
 +    * FV physical visoon 
 +    * P-FV up -) P-NV down 
 +    * P-NV up -) P-FV down 
 +    * P-NV up peaks (value decrease) = "hypnagogic images" 
 +    * P-NV large down peaks or P-FV large up pekas = eye movement of focus to FV
 +    * orange arrows area = behaind the eyelids backnes in this cases rednes (focus to FV)    
 +    * orange points P-FV up and P-NV up = other sense influence,thoughts,integration .. (need more research)   
 +=== Pasive realx - morning ===
 +  *red dot = sneeze
 +  *pimk dots = large eye movements (also possibe focus to FV without the movement but less probable )   
 +=== Active realx - focus to breath and heart beat ===
 +   * evening 20:30
 +   * hard to focus
 +   * chaotic thoughts 
 +   * cognitive overload (tetris effect)about 6h watching new TV serial , this increase base P-NV 
 +   * P-NV higher than 3 after 15s (probably due the cognitive overload)
 +   * T 2,75 P-NV and P-FV decrease probaly due focus to other senses or thought 
 +   * T pink line = auto eyes open , few times durring the session my eyes slightly open by themselves  
 +   * T green line = dream scene , no loger flashes of image but scene (may be caused by visualization , automatic asociation about thought tema , or themselfs)
 +     visualized ones have higer P than asociation , asociation  higher than automatic (in similar time in one session )
 +   * P-FV below 3 = very important moment mean physical vision is turn off and allow NV disinhibition    
 +=== Active relax - focus to breath and heart beat - test 2 ===
 +   * same day evening 22.00
 +   * more awake than in previous test but less images 
 +   * P-NV higher than 3 after about 18s (probably due the cognitive overload)
 +   * T 0:18-0:45 asociation created images i remeber few names from serial 
 +   * T 1:08 P-FV and P-FV go down probably because start focusing to breath and heart rate (probably increase of propriorecoption P )
 +   * T 4:45 P-FV uder 3
 +   * T 6:22 - 9:00 probably proprioreception effect
 +   * gereen line above T 12:30 active vizualization of flight on dragon (scene form serial) ,2 scenes before caused by mix of visualization nad asociation (more  asociation)     
 +=== Some test (second part of larger test unfinished )===
 +  * before  drawing 12 min of relax and eye open 
 +  * {{http://brmlab.cz/_media/project/brain_hacking/some_test.jpg}}
 +  * T around 1:45 green line = vizualization -) images
 +  * T around 2:00 yellow line = after vizualization automatic asociation nad scene with lower P
 +=== Realx - Breath  - morning ===
 +  * 9:30h
 +  *shorter inter flash intreval than in other test (morning ?)
 +  *P-NV stabil larger than 3 1:40
 +  *P-FV below 3 : 5:00
 +  *T red line = behaind the eyelids blacknes in this cause redness in this sessoin cause problems (too much light in the room)  
 +  *T 10-11 focus to FV redness
 +  *viz = active vizualization 
 +=== EOG biofeedback ===
 +  *audio EOG SD1s amplitude(4,5uV+) to volume  as feedback
 +=== Realx - guided 7min ===
 +  * guided relaxation
 +  *FP physical proprioreception
 +  *P-FP allow distiguish between eye movement and focus without movement  
 +  *T 10:45 - 11:30 increase of P-FP : focus to body ? cause P-FV go below 3 ? --) need more tests 
 +TO DO --)increase vertical resolution and add more senses
 +==== 2.6 ====
 +  *Fleeting vs Fully-Formed Hypnagogic Images.
 + 'fleeting' vs. 'fully-formed' images. These
 +two image types are distinguishable by several qualities, including the following.
 +  * 1)
 +Temporal. Fleeting images tend to occur during the first stages of a self-observa tion trial, before a fully-formed image has arisen. 
 +  * 2)Duration.Fleeting images are brief; they appear and disappear in an instant, often they are not detectible during
 +self-observation without a firmly established intent. Fully-formed images are more enduring and thus more easily observed and recalled. 
 +  *3)Simplicity
 +. Unlike a fully-formed hypnagogic image, a fleeting image may occur in only a single sensory modality. It may consist of nothing more
 +than an isolated flash of visual scenery, a single fragment of spoken voice, or a brief moment of kinesthetic sensation. Fully-formed images may be comprised of
 +all three modalities. 
 +  *4)
 +. Because they are brief, simple and outside the range of most observers' waking and dreaming experiences, fleeting images may easily defy
 +verbal description. Momentary kinesthetic sensations
 +are particularly difficult to describe because there is a lack of a commonly accepted vocabulary for the domain of
 +kinesthetic experience. Fully-formed images more nearly resemble nocturnal dreams and are similarly more amenable to description. To characterize the nature of fleeting images adequately, an observer must often advance either metaphoric discourse or completely novel terminology. 
 +  *5)
 +Association with sleep onset feelings. As shown
 +in Table 2, fleeting images are often completely inseparable from subtle sleepiness feelings, as if the images were
 +an integral part of sleepiness itself. Although sleep onset feelings may also accompany fully-formed images, in
 +these cases they are often not obvious. Their ephemeral nature may be masked by the dramatic complexity of
 +more fully-formed multimodal imagery
 +<note>5)depend of used method </note>
 +<note>my method use more phases of hypnagogic imaginery (in part : Image properties) </note>
 +  *Phasic neuromuscular events (NMEs) which regul
 +arly accompany hypnagogic images, such as limb jerks,
 +were found to be isomorphic with some imagery content. In Study 1 (fully-formed images), phasic NMEs were
 +found to accompany 56.7% of images. In Study 2 (fleeting images), this proportion was only 16.7%. However,
 +the proportions of phasic NME types for the two classes of
 +imagery were strikingly similar: head tilts (61.5% v.
 +56.4%), leg jerks (23.1% v. 23.1%), arm jerks (7.7% v.
 +15.4%) and whole body jerks (7.7% v. 5.1%) respectively.
 +It seems likely that many NMEs accompanying fleeting images
 +were too subtle or transient to be detected by this
 +self-observation technique, a possibility supported by physiological recordings, such as those in Study 3. 
 +  * Although the duration of the pre-image interval was
 +variable, the pattern of EEG changes was similar
 +from image to image. These findings suggest that the production of hypnagogic imagery for this observer may
 +have a more or less invariant underlying physiological pattern. The relationship of this pattern to that of the
 +previously-described phenomenological events remains tobe clarified. However, it seems likely that the observed
 +in EEG activity correspond to periods of activation and deactivation of hypnagogic imagery processes
 + * In most cases, cessation of eye
 +activity was followed by a diminution in alpha activity and EEG amplitude across most channels and an increase
 +in the prevalence of other frequency bands.
 +==== 4.6 ====
 +Lucid Dream by use of multisensory integration NV scene H 2-3 stabilized by sense of touch in legs to H5.
 +Some route in forest near water i look at feet and focus to sense of touch (to grass , groud)it takes bout 20s to stabilize to H5.LD have about 5 parts whn drem destabilize i stabilze it again sometimes i almoust lost sense of vison.
 + * i have EEG recording form Fp1 and T5 somwhere in it is LD recorded 
 +  *CH1 Fp1 A1
 +  *CH2 T5  A1
 +  *GND   A2
 +  *relaxation on back  
 +  *40 move head to right side (stil on back)
 +  *50 move to side 
 +  *65 big change (62:40-65:00)
 +  *65-75 start of LD  (not enought data and experinece to determine if start 65 or 75min) ,(automatic drawing show strat 65 but information show only NV without inromation about R and other importatnt to detemine LD) 
 +Recording for Automatic drawing was turn OF i try reconstrut some inromation from memory but resutl gave many gaps and i doun tknou how reaiable it is .
 +Sleep spindle
 +  *Sleep spindles (sometimes referred to as "sigma bands" or "sigma waves") may represent periods where the brain is inhibiting processing to keep the sleeper in a tranquil state. Along with K-complexes they are defining characteristics of, and indicate the onset of, stage 2 sleep. They are often tapered at both ends and frequently seen over the frontal and central head regions. They may or may not be synchronous, but they should be symmetrical and bilateral.
 +  *During sleep these spindles are seen in the brain as a burst of activity immediately following muscle twitching.
 +  *The greatest spindle activity occurs at the beginning and the end of the non-REM portion of the sleep cycle.
 +  * There are also slow spindles in the range of 11 - 13 Hz that are also associated with increased activity in the superior frontal gyrus, and fast spindles in the range of 13 - 15 Hz that are associated with recruitment of sensorimotor processing cortical regions, as well as recruitment of the mesial frontal cortex and hippocampus. What do these spindles mean? Ongoing research hopes to illuminate their function.
 +  *Recent research by Matthew Walker and his research team at the University of California Berkley shows that sleep spindles are associated with refreshment of our ability to learn. This research showed that the greater the number of sleep spindles produced by napping participants, the more they were refreshed to perform on a learning task. Further, the brain areas most involved were the hippocampus and the prefrontal cortex. These are areas that are critical for learning.
 +  *The incidence of spindles is reported to peak at a frequency of 0.2-0.3 Hz
 +  *The incidence of spindles has considerable variance (1±40 s inter-spindle intervals in humans). Also variable is their topographic prevalence in the brain, their time of appearance in sleep stages, their association to other EEG landmarks (like K-complexes) and their dependence on drugs. All these suggest that spindles do not constitute a unique and/or uniform phenomenon. 
 +  *The association of spindles the slow cortical oscillation (~0.75 Hz) [12] is proposed to be causative in the sense that this oscillation which is supposedly generated within neocortical networks, synchronizes neuronal activity into generalized down-states (hyperpolarization) of global neuronal silence and subsequent up-states (depolarization) of increased wake-like neuronal firing. With the beginning of the latter cortico-thalamic volleys are proposed to drive the generation of spindle activity. 
 +  *Such a strong and synchronous input may discharge reticular cells directly or indirectly and thus could serve as the initiator of sleep spindles [2]). In a recent study the incidence of spindles immediately following K-complexes was between 65-70%
 +  *However in this study neither the probability of appearance nor the power of spindles correlated to the amplitude or any other feature of the K-complexes that preceded them. When K-complexes appeared spontaneously after the start of a sporadic spindle, the spindles were invariably shut down for the duration of the K-complex, usually being replaced by a short lived oscillation in the high theta frequency band. Also the spindles appearing immediately after a K-complex had invariably faster spectral frequency than the sporadic spindles. Such findings suggest that the association of K-complexes with spindles is strong but may be due to a common trigger rather than a causative interaction.
 +  *Animal and human studies show that spindles are sleep maintaining events [65-66] that block the transfer of sensory information to the cerebral cortex during sleep [64], thus preventing sleep-interrupting arousals.
 +  *While TC neurons are hyperpolarized and engaged in this bursting mode, sensory afferents are expectedly prevented from reaching the cortex, resulting in almost complete deafferentation, except for very strong or alarming stimuli [65].
 +  *The recent observation (described in 2.2.) that spindles are invariably shut down for the duration of the K-complex and they appear right after at increased spectral frequency [71] support a role of spindles in preventing stimuli (which triggered the K-complex) to reach consciousness.
 +  *Anesthetics lead also to loss of consciousness - in the sense of turning the subjects oblivious to their environment - with different mechanisms depending on the drug used. One mechanism they partly share with natural sleep is apparently the production of spindles in a similar way, as several anesthetics hyperpolarize TC neurons (by activating 2PK channels and/or by potentiating GABA receptors) and halothane induced spindles are antagonized when carbachol is injected into the pontine reticular formation [74]. Spindling then causes a decorrelation between sensory input to TC neurons and these neurons’ output to the cortex, thus contributing to the loss of consciousness. 
 +  *http://www.intechopen.com/books/advances-in-clinical-neurophysiology/sleep-spindles-as-a-biomarker-of-brain-function-and-plasticity
 +K complexes 
 +  *are large waves that stand out from the background and often occur in response to environmental stimuli such as sounds in the bedroom.
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